Preliminary experiments reveal conolidine may inhibit particular ion channels, decreasing neuronal excitability and restricting discomfort alerts. This mechanism is particularly suitable in neuropathic ache, the place abnormal signaling results in persistent irritation. In addition, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to agony perception. ... https://anthonyk823zzb0.newsbloger.com/profile